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Table 2 The distribution of APOE genotyping in all studied cases

From: The independent association between 25 (OH) vitamin D deficiency, HOMA-IR, and lipid profile with APOE genotyping in obese cases with and without T2DM

APOE genotyping & alleles

Control group.

N = 100

Obese cases

N = 100

obese cases with T2DM

N = 100

P- value

Control Vs

obese cases

Control Vs obese cases withT2DM

Obese cases without and with T2DM

E3/E3 (ref)

19 (19%)

21 (21%)

13 (13%)

0.055

< 0.001

0.005

E2/E2

0

3 (3%)

7 (7%)

E2/E3

15 (15%)

26 (26%)

10 (10%)

E2/E4

20 (20%)

11 (11%)

22 (22%)

E3/E4

46 (46%)

39 (39%)

31 (31%)

E4/E4

0

0

17 (17%)

E3 (ref)

99 (45.5%)

79 (39.5%)

67 (33.5%)

< 0.001

0.005

< 0.001

E2

35 (17.5%)

71 (35.5%)

46 (23%)

E4

66 (33%)

50 (25%)

87 (43.5%)